During gastrovirusnoy infections should avoid consumption of certain products

True. In the case of gastroenteritis normal appetite may return within a few hours after vomiting. But this does not mean that you can eat as before, flavoring dishes spicy sauces and fatty sauces that irritate the stomach and intestines.

Among the prohibited during this period: dairy products, citrus fruits, hot and spicy dishes that are rich in fats, all raw fruits except bananas for Bananas: the benefits and harms health . They should be avoided, at least forty-eight hours after the complete termination of emesis. And do not forget about the need for copious drinking.

To prevent the disease should eat only freshly washed fruits and vegetables

Viral gastroenteritis is very easy to spread, mainly through the hands and other tactile contact with the patient. If fruits and vegetables were in contact with patients, they should be well cleaned and dried before it is. But the virus in the environment is unstable, and a few minutes later, he dies.

Remember that washing your hands - it's a habit that should be a skill, and it is necessary to resort frequently, after contact with the environment, but making sure that the skin does not lose precious moisture. This is especially important during epidemics and in contact with sick people. Children from an early age should be accustomed to washing hands with soap.

Products that have expired - the sources of virus

Not true. The infection has nothing to do with the consumption of stale food, as the virus outside the body is not able to survive and dies in a few minutes. Therefore, no risk.

Gastroenteritis, which occurs when the consumption of spoiled food, a bacterial origin - bacteria develop in the product, and then enter the body and cause disease. To protect themselves from this disease, it is necessary to monitor temperature conditions in which food is stored, paying attention to the expiration date.

Infection with the virus that causes gastroenteritis is from a sick person. But there should be careful, because it happens that the patient does not feel symptoms, but already is contagious.

Causes of liver steatosis

Steatosis (fatty liver) - a disease which resulted in impaired lipid metabolism in liver cells (hepatocytes) resulting in a sharp obesity liver cells and their death without inflammation.

Steatosis may be acute or chronic. Acute fatty liver often develops in cases of poisoning by phosphorus, arsenic, ethyl alcohol, some plants under the influence of drugs (such as tetracycline in large doses). It can be observed in the last weeks of pregnancy, due to protein deficiency the mother's body (all of the protein goes to the fetus), and vomiting associated with toxemia of pregnancy.

Chronic steatosis often occurs against the backdrop of a long continuous alcohol abuse. Also relevant are such long-occurring diseases such as diabetes mellitus Diabetes - threatening and incurable disease   (more common in the elderly), general obesity, lack of proteins and vitamins in the diet, chronic intoxication (poisoning) organophosphorus compounds and bacterial toxins (chronic inflammation in other organs).

If the main reason for alcoholic fatty liver is alcohol abuse, why develop non-alcoholic fatty liver, it is not yet clear. However, the factors that increase the risk of developing this disorder, among them - family history, high cholesterol or triglycerides, diabetes, and prediabetes (insulin resistance). Possible causes of fatty liver are also:

• Oxidative stress How to beat stress? Create an oasis Which causes damage to the liver cells.
• Toxic inflammatory proteins from fat, liver and other cells.
• The death of liver cells (apoptosis).
• Medications.
• Viral hepatitis.
• Autoimmune liver disease or congenital.
• Rapid weight gain.
• Malnutrition.
• Malnutrition.

Recent studies give reason to assume that the uncontrolled proliferation of bacteria in the small intestine and some other changes in the intestines are also associated with the development of non-alcoholic fatty liver.

Insulin resistance

It was once thought that obesity - it's just the accumulation of fat in the body, and nothing more. Adipose tissue is considered inert - it was assumed that she was just a strategic reserve of the body in case of famine, and by itself it is not active and can not communicate with other tissues. For modern science it is clear that adipose tissue has a high metabolic activity, and may affect different tissues and structures throughout the body. It becomes most metabolically active when the body is deposited a large amount of fat - in excess weight and, particularly, obesity: adipose tissue leads to the production of various hormones and proteins that are released into the blood and affect the cells of other tissues. One result of the activity of these hormones and proteins may be the development of insulin resistance.

Insulin - a condition in which the cells of the body can not adequately respond to insulin, a hormone produced by the pancreas. Insulin plays a critical role in the absorption of glucose from the blood cells of the body. At first, the pancreas tries to compensate for insensitivity to insulin The principles of action of insulin - the science of saving lives   generation of large amounts of the hormone, but in the end, there comes a moment when it is no longer able to secrete insulin in sufficient quantities. As a consequence, the body cells insufficiently fed glucose, and it accumulates in the blood - a condition known as diabetes. Because glucose is the major source of energy for cells and allows them to perform the functions for which they are intended, lack of glucose inevitably leads to changes in cell function.

As a result of these processes, liver cells, like many other cells in the body become insulin resistance and metabolic processes therein, including processing on fat, are subject to change. Hepatic tissue cells begin to absorb more fatty acids from blood, where the latter are present in excess. Then, in the liver fatty acid is converted to fat in the form in which it is usually stored in the body. At the same time, the ability to withdraw excess fat liver is broken and its concentration increases gradually. Besides, the liver continues to produce oil and getting it from food. The consequence of this is the development of fatty liver.

It should be noted that this violation is not found in all patients with diabetes or obesity - probably there are some contributing factors that determine, in an individual patient will develop steatosis, or not.

Other possible causes of fatty liver liver:

• Metabolic syndrome. This disorder can lead to obesity, high triglyceride levels, decrease the level of HDL-cholesterol, hypertension and diabetes. Many of these factors, as discussed above, are associated with the development of fatty liver;
• Hepatitis C;
• Taking certain medications, such as corticosteroids, tamoxifen and methotrexate;
• Abetalipoproteinemia.

The symptoms of acute fatty liver

Acute fatty liver begins suddenly and proceeds rapidly. This is manifested by diarrhea, jaundice and hemorrhagic (bleeding in the form of small blood vessels), rash on the skin. The liver is usually not increased or increased slightly.

The disease can be complicated by hepatic coma, severe renal failure, bleeding.

Acute hepatic steatosis to a pregnant woman - serious diagnosis that requires immediate hospitalization and active treatment. In mild cases, appropriate treatment with proper structure and function of the liver can recover. Sometimes during it becomes chronic.

Symptoms of chronic fatty liver

Chronic steatosis begins unnoticed, for it is characterized by enlargement of the liver, sometimes significantly. At the touch of the liver is smooth and thick. Often there is a general weakness, dull pain in the right upper quadrant, diarrhea, light yellow skin. Sometimes the disease goes unnoticed, disguised manifestations of the underlying disease that caused steatosis (eg, diabetes).

The course of chronic hepatosis relatively favorable, with full restoration of the structure and function of the liver. But chronic steatosis is reversible, if not started restructuring of the liver. The disease may progress to chronic hepatitis Hepatitis - the scourge of our time   (inflammation), alcohol abuse, after an infection and so on. The end result is often chronic hepatitis cirrhosis (replacement of renal tissue connecting with persistent decline in liver function).